Max in WT synaptoneurosomes, suggesting that Src signaling can be downregulated in KI synapses. On the flip side, our capability to rescue SERT function in KI midbrain synaptoneurosomes via the inhibition of FAK suggests elevated FAK signaling downstream in the Pro32Pro33 mutant, as confirmed by amplified pFAK localization in five-HT https://alexanderb107fqd9.blogmazing.com/profile